Cauda equina syndrome
Editor-In-Chief: Prab R Tumpati, MD
Obesity, Sleep & Internal medicine
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| Cauda equina syndrome | |
|---|---|
| Synonyms | N/A |
| Pronounce | N/A |
| Specialty | N/A |
| Symptoms | Low back pain, sciatica, saddle anesthesia, bladder dysfunction, bowel dysfunction, sexual dysfunction |
| Complications | Permanent paralysis, urinary incontinence, fecal incontinence |
| Onset | Sudden or gradual |
| Duration | Variable |
| Types | N/A |
| Causes | Herniated disc, spinal stenosis, tumors, trauma, infection |
| Risks | Spinal surgery, spinal anesthesia, lumbar puncture |
| Diagnosis | MRI, CT scan, myelogram |
| Differential diagnosis | Conus medullaris syndrome, lumbar spinal stenosis, peripheral neuropathy |
| Prevention | N/A |
| Treatment | Surgical decompression, corticosteroids, pain management |
| Medication | N/A |
| Prognosis | Better with early treatment |
| Frequency | Rare |
| Deaths | N/A |
A serious neurological condition affecting the cauda equina nerves
Cauda Equina Syndrome
Cauda equina syndrome (CES) is a serious neurological condition that occurs when the bundle of nerves at the end of the spinal cord, known as the cauda equina, is compressed. This compression can lead to a range of symptoms, including severe back pain, loss of sensation, and motor weakness in the lower body. It is considered a medical emergency that requires prompt diagnosis and treatment to prevent permanent damage.
Anatomy
The cauda equina is a bundle of spinal nerves and nerve roots that originate from the lower end of the spinal cord. These nerves resemble a horse's tail, which is how the condition gets its name ("cauda equina" is Latin for "horse's tail"). The cauda equina is responsible for sending and receiving messages to and from the lower limbs and pelvic organs.
Causes
Cauda equina syndrome can be caused by several conditions that lead to compression of the cauda equina nerves. Common causes include:
- Herniated disc: A herniated or slipped disc in the lumbar region can press on the cauda equina.
- Spinal stenosis: Narrowing of the spinal canal can compress the nerves.
- Tumors: Growths in the spinal canal can exert pressure on the cauda equina.
- Trauma: Injuries to the lower back can lead to nerve compression.
- Infections: Infections such as spinal epidural abscess can cause swelling and compression.
- Inflammatory conditions: Conditions like ankylosing spondylitis can lead to inflammation and compression.
Symptoms
The symptoms of cauda equina syndrome can vary but often include:
- Severe lower back pain
- Sciatica (pain radiating down the leg)
- Numbness or tingling in the "saddle" area (inner thighs, buttocks, and back of the legs)
- Weakness in the legs
- Loss of reflexes in the lower extremities
- Bladder and bowel dysfunction, such as urinary retention or incontinence
Diagnosis
Diagnosis of cauda equina syndrome involves a combination of clinical evaluation and imaging studies. A thorough neurological examination is essential to assess motor and sensory function. Imaging studies such as MRI or CT scan are used to visualize the spinal canal and identify the cause of compression.
Treatment
Cauda equina syndrome is a surgical emergency. The primary treatment is surgical decompression to relieve pressure on the cauda equina nerves. The timing of surgery is critical; early intervention, typically within 48 hours of symptom onset, is associated with better outcomes. Postoperative care may include physical therapy and rehabilitation to address any residual deficits.
Prognosis
The prognosis for cauda equina syndrome depends on the severity and duration of nerve compression before treatment. Early diagnosis and treatment are crucial for minimizing permanent damage. Some patients may experience persistent symptoms such as weakness or bladder dysfunction, even after surgery.
Prevention
While not all cases of cauda equina syndrome can be prevented, maintaining good spinal health can reduce the risk. This includes regular exercise, proper lifting techniques, and managing conditions like osteoporosis and arthritis.
See also
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Contributors: Prab R. Tumpati, MD
