Anetoderma
(Redirected from Schweninger–Buzzi anetoderma)
Anetoderma | |
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Synonyms | Macular atrophy |
Pronounce | N/A |
Specialty | N/A |
Symptoms | Skin lesions, atrophy |
Complications | None typically |
Onset | Usually in adolescence or early adulthood |
Duration | Chronic |
Types | N/A |
Causes | Unknown, possibly autoimmune or genetic factors |
Risks | Family history, autoimmune disease |
Diagnosis | Clinical diagnosis, skin biopsy |
Differential diagnosis | Pseudoxanthoma elasticum, mid-dermal elastolysis, cutaneous mastocytosis |
Prevention | N/A |
Treatment | No definitive treatment, options include topical and systemic therapies |
Medication | N/A |
Prognosis | Benign, cosmetic concern |
Frequency | Rare |
Deaths | N/A |
Anetoderma is a localized skin condition with its characteristic button hole type leisons.
Cause
It is due to abnormal laxity of the skin with herniation or outpouching resulting from abnormal dermal elastic tissue.
Types
- Primary anetoderma
- Jadassohn–Pellizzari anetoderma
- Schweninger–Buzzi anetoderma
- Secondary anetoderma
- Familial anetoderma
Secondary anetoderma
Any of the following conditions can cause secondary anetoderma
- Infections such as chickenpox, syphilis
- Tumours such as pilomatrixoma, plasmacytoma, schwannoma, cutaneous B-cell lymphoma
- Infantile haemangioma
- Inflammatory skin conditions such as acne, granuloma annulare, lupus profundus
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